
Monday, March 16, 2009
Laser Gun : can kill the mosquitoes

Saturday, March 14, 2009
Nepal : a photographic tour
THe best tourist destination .

Country of natural beauty.

End of the 240 years of History of Monarchy .

Initialization of new era in Nepali history .

Living goddess "Kumari"

This reminds the 12 years long insurgency in Nepal.

Kathmandu; the city of Temples.

Political instability, which is very common in Nepal.

Slums of Nepal.

In The day of color "Holi".
Friday, March 13, 2009
laser

The word laser originated as an acronym for light amplification by stimulated emission of radiation. The word light in this phrase is used in the broader sense, referring to electromagnetic radiation of any frequency, not just that in the visible spectrum. Hence there are infrared lasers, ultraviolet lasers, X-ray lasers, etc. Because the microwave equivalent of the laser, the maser, was developed first, devices that emit microwave and radio frequencies are usually called masers. In early literature, particularly from researchers at Bell Telephone Laboratories, the laser was often called the optical maser. This usage has since become uncommon, and as of 1998 even Bell Labs uses the term laser.
The back-formed verb to lase means "to produce laser light" or "to apply laser light to". The word "laser" is sometimes used to describe other non-light technologies. For example, a source of atoms in a coherent state is called an "atom laser".
Chemical lasers
Chemical lasers are powered by a chemical reaction, and can achieve high powers in continuous operation. For example, in the Hydrogen fluoride laser (2700-2900 nm) and the Deuterium fluoride laser (3800 nm) the reaction is the combination of hydrogen or deuterium gas with combustion products of ethylene in nitrogen trifluoride. They were invented by George C. Pimentel.
C.V Raman

Chandrashekhara Venkata Raman (C.V.Rama) is a physicist and nobel laureate in Physics in 1930 for his work on "Raman Effect",Scattering of light. Raman entered Presidency College, Madras, in 1902, and in 1904 gained his B.Sc., winning the first place and the gold medal in physics. In 1907 he gained his M.Sc., obtaining the highest distinctions. He joined the Indian Finance Department as an Assistant Accountant General in Calcutta.February 28, 1928, through his experiments on the scattering of light, he discovered the Raman effect.Read more
On It was instantly clear that this discovery was an important one. It gave further proof of the quantum nature of light. Raman spectroscopy came to be based on this phenomenon. Rutherford referred to it his presidential address to the Royal Society in 1929. Raman was president of the 16th session of the Indian Science Congress in 1929. He was conferred a knighthood, and medals and honorary doctorates by various universities. Raman was confident of winning the Nobel Prize in Physics as well, and was disappointed when the Nobel Prize went to Richardson in 1928 and to de Broglie in 1929. He was so confident of winning the prize in 1930 that he booked tickets in July, even though the awards were to be announced in November, and would scan each day's newspaper for announcement of the prize, tossing it away if it did not carry the news. He did eventually win the 1930 Nobel Prize in Physics "for his work on the scattering of light and for the discovery of the effect named after him". He was the first Asian and first non-White to get any Nobel Prize in Science. Before him Gurudev Rabindranath Tagore (also Indian) had received the Nobel Prize for Literature.In 1954 he was awarded the Bharat Ratna. He was also awarded the Lenin Peace Prize in 1957.
India celebrates National Science Day on 28 February of every year to commemorate the discovery of the Raman effect in 1928
Hollow-Cathode Lamp

The one of the most common source for atomic absorption measurements is the hollow cathode lamp. Usually these kind of lamps consists of a tungsten anode and a cylindrical cathode sealed in a glass tube filled with neon or argon at the pressure of 1 to 5 torr. The cathode is constructed of the metal whose spectrum is desired or serves to support a layer of that metal .
Ionization occurs when a potential difference on the order of 300V is applied across the electrodes. The efficiency of the hollow cathode lamp depends on its geometry and the operating voltage. High voltages,and thus high currents, leads to greater intensities . READ MORE
This advantage is offset somewhat by an increase in Doppler broadening of the emission lines from the lamp. Hollow-cathode lamps are often used as source in AFS. In this application the lamps are pulsed with a duty cycle of 1% to 10% and peak current of 0.1 to 1 A, which increase their peak radiance by a factor of 10 to 100 relative to the steady state radiance of DC operation . A variety of hollow cathode lamps is available commercially . The cathodes of some consists of a mixture of several metals ;such lamps permit the determination fo more than a single element.see more in wikipedia
Thursday, March 12, 2009
X-ray Spectrometry


X-rays are short-wavelength electromagnetic radiation by the deceleration of high-energy electrons or by electronic transitions of electrons in the inner orbitals or atoms.The wavelength range of X-rays is from about 10e-5A to 100A;conventional X-ray spectroscopy is,however ,highly confined to the region of about 0.1A to 25A (1A=0.1nm=10e-10).
Emission of X-rays
For analytical purposes,X-rays are generated in four ways. 1.by bombardment of a metal target with a beam of high energy electrons ,2) by exposure of a substance to a primary beam of X-rays to generate a secondary beam of X-ray fluorescence,3)by use of a radioactive source whose decay process results in X-ray emissions,and 4)from a synchrotron radiation source. Only limited laboratory in US allows to produce X rays from synchrotron radiation.Contd.
Type rest of the post here
Monday, March 9, 2009
How To Send SMS without any Cost?

Yes ! you can send the SMS to your friend without paying any money to corresponding phone company in United States. Just open your web browser , sign in your any email and you need to type the receiver cell no. followed by the email address of corresponding mobile company service providers.This is only for mobile companies with in the United States. CLICK HERE FOR MORE
T-Mobile: phonenumber@tmomail.net
Virgin Mobile: phonenumber@vmobl.com
Cingular: phonenumber@cingularme.com
Sprint: phonenumber@messaging.sprintpcs.com
Verizon: phonenumber@vtext.com
Nextel: phonenumber@messaging.nextel.com
Where phonenumber corresponds to the receiver cell number.
OK Have fun by sending the SMS to your friends,loved one without paying any money.
Sunday, March 8, 2009
Einstein Brain : What is the difference?

Albert Einstein
One sign of the lack of faith in the future progress of technology and the poor acceptance of the neurological basis for mind is the way in which our society treats the “post-mortem” human brain.
In some cases, the brains of those whom modern medicine cannot help are removed after cardiopulmonary arrest and donated (by the permission of the patient or the family) CLICK HERE TO READ MORE fo
Top 10 reasons to smile : )

Bisphenol A (BPA) : what is it? Q and A

Bisphenol A, commonly abbreviated as BPA, is an organic compound with two phenol functional groups. It is a difunctional building block of several important plastics and plastic additives. With an annual production of 2–3 million metric tonnes, it is an importantmonomer in the production of polycarbonate.
Suspected of being hazardous to humans since the 1930s, concerns about the use of bisphenol A in consumer products grabbed headlines in 2008 when several governments issued reports questioning its safety, and some retailers pulled products made from it off their shelves.Click here for more...
What is BPA?
Bisphenol A (BPA) is an industrial chemical used to make polycarbonate plastic resins, epoxy resins, and other products.
Bisphenol A (BPA) is a chemical building block that is used primarily to make polycarbonate plastic and epoxy resins. Polycarbonate plastic is a lightweight, high-performance plastic that possesses a unique balance of toughness, optical clarity, high heat resistance, and excellent electrical resistance. Because of these attributes, polycarbonate is used in a wide variety of common products including digital media (e.g., CDs, DVDs), electrical and electronic equipment, automobiles, sports safety equipment, reusable food anddrink containers , and many other products.
BPA is also used in the production of epoxy resins. Epoxy resins have many uses including engineering applications such as electrical laminates for printed circuit boards, composites, paints and adhesives, as well as in a variety of protective coatings. Cured epoxy resins are inert materials used as protective liners in metal cans to maintain the quality of canned foods and beverages, and have achieved wide acceptance for use as protective coatings because of their exceptional combination of toughness, adhesion, formability, and chemical resistance.
In 2002, approximately 2.8 million tons of bisphenol A (BPA) was produced globally (Source: Chemical Market Associates, Inc. (CMAI)). Most BPA is used to make polycarbonate plastic and epoxy resins.
Has BPA been tested for safety?
Yes. Bisphenol A (BPA) is one of the most extensively tested materials in use today. BPA has been safely used in consumer products and researched and studied for over 40 years. The weight of scientific evidence clearly supports the safety of BPA and provides strong reassurance that there is no basis for human health concerns from exposure to BPA.
Does BPA pose a risk to human health?
Safety assessments of bisphenol A (BPA) conclude that the potential human exposure to BPA from polycarbonate plastics and epoxy resins is more than 400 times lower than the safe level of BPA set by the U.S. Environmental Protection Agency. This minimal level of exposure to BPA poses no known risk to human health.
The use of polycarbonate plastic and epoxy resins for food contact applications has been and continues to be recognized as safe by the U.S. Food and Drug Administration, the European Commission's Scientific Committee on Food, the United Kingdom Food Standards Agency, and otherregulatory agencies worldwide.
Am I exposed to BPA from polycarbonate plastics?
Researchers from government agencies, academia, and industry worldwide have studied the potential for bisphenol A (BPA) to migrate from polycarbonate products into foods and beverages. These studies consistently show that the potential migration of BPA into food is extremely low, generally less than 5 parts per billion under conditions typical for uses of polycarbonate products. At this level, a consumer would have to ingest more than 1,300 pounds of food and beverages in contact with polycarbonate every day for an entire lifetime to exceed the safe level of BPA set by the U.S. Environmental Protection Agency. Consequently, human exposure to BPA from polycarbonate plastics is minimal and poses no known health risk.
The use of polycarbonate plastic for food contact applications continues to be recognized as safe by the U.S. Food and Drug Administration, the European Commission Scientific Committee on Food, the United Kingdom Food Standards Agency, the Japan Ministry for Health and Welfare and other regulatory authorities worldwide.
Am I exposed to BPA from can linings?
Government and industry researchers have reported that bisphenol A (BPA) is generally not detected in canned beverages and only extremely low levels (generally less than 37 parts per billion) of BPA have been reported to migrate into some canned foods. At these levels, a consumer would have to ingest more than 500 pounds of canned food and beverages every day for an entire lifetime to exceed the safe level of BPA set by the U.S. Environmental Protection Agency. Consequently, human exposure to BPA from can coatings is minimal and poses no known health risk.
Can coatings continue to be recognized as safe by the U.S. Food and Drug Administration, the U.K. Food Standards Agency, the EU Scientific Committee on Food and other government bodies worldwide.
Does BPA leach out of dental sealants?
Several studies have reported that trace levels of bisphenol A (BPA) may be released from certain dental sealants, but only during a short time period immediately after application of the sealant. In addition, the highest level of BPA exposure reported from dental sealants is more than 50,000 times lower than levels shown to cause toxicity in animal studies. Based on these findings, human exposure to BPA fromdental sealants is minimal and poses no known health risk.
What about claims that very low-dose exposure to BPA has resulted in reproductive effects in laboratory animals?
The low-dose hypothesis for bisphenol A (BPA) has been thoroughly tested with a series of comprehensive, carefully conducted studies. This research includes definitive large-scale studies as well as studies aimed at replicating the results of studies reporting low-dose effects. The consistent lack of low-dose effects found in these studies demonstrates that the low-dose hypothesis is not valid.
The weight of scientific evidence clearly supports the safety of BPA and provides strong reassurance that there is no basis for human health concerns from exposure to low doses of BPA.
No. According to U.S. Environmental Protection Agency records, bisphenol A (BPA) has not been used as an inert ingredient in pesticide products in the U.S. since at least 1994. BPA's status as an (inert) pesticide ingredient was recently rescinded by EPA, according to the June 11, 1999 Federal Register. According to the notice, BPA was removed from a listing of approved inert substances because it was not in use as an additive.
Is BPA found in the environment?
Although the vast majority of bisphenol A (BPA) is converted at manufacturing sites into products, low-level releases of BPA to the environment are possible. Government researchers have reported that, when detected at all, BPA is found in water at levels generally well below 1 part per billion.
Extensive testing and environmental monitoring shows that BPA is rapidly biodegraded in the environment. The weight of scientific evidence shows that the trace amounts of BPA that are sometimes detected in waterways pose no risk to the environment.
Does BPA adversely impact aquatic organisms?
The weight of evidence from numerous validated studies demonstrates the trace levels of BPA that have been detected in the environment are far below the level at which adverse effects on aquatic organisms would be expected. Bisphenol A (BPA) does not accumulate in aquatic organisms to any appreciable extent and is not classified as bioaccumulative by the U.S. Environmental Protection Agency.
Is there an alternative to BPA in consumer products?
Bisphenol A (BPA) is used primarily to make polycarbonate plastic and epoxy resins, and is integral to the manufacture of both materials. Both polycarbonate and epoxy are unique and versatile materials that out-perform other materials in a broad range of end-uses providing consumers with a unique range of properties not available in other materials. Polycarbonate plastic is a lightweight, high-performance plastic that possesses a unique balance of toughness, optical clarity, high heat resistance, and excellent electrical resistance that make it ideal for a wide variety of applications. Epoxy resins are inert materials that have achieved wide acceptance for use as protective coatings and other applications because of their exceptional combination of toughness, adhesion, formability, and chemical resistance. Epoxy can coatings are essential to protecting food and beverages from contamination.
BPA is one of the most extensively tested materials in use today; its safety has been studied for more than 40 years. The extensive safety data that exist for BPA show that consumer products made with BPA are safe for their intended use and pose no known risks to human health. The U.S. FDA and international agencies charged to protect public health fully support the use of these materials.
Saturday, March 7, 2009
How to know the traffic rank of the blog,website?

Chemical and Physical properties Used in Instrumental Methods

Characteristic Properties And Instrumental methods
Carbamazepine : Mood Stablizing drug

Carbamazepine (CBZ) is an anticonvulsant and mood stabilizing drug used primarily in the treatment of epilepsy and bipolar disorder. It is also used to treat ADD, ADHD, schizophrenia, phantom limb syndrome, paroxysmal extreme pain disorder, and trigeminal neuralgia.
Trade names
Carbamazepine has been sold under the names Tegretol, Biston, Calepsin, Carbatrol, Epitol, Equetro, Finlepsin, Sirtal, Stazepine, Telesmin,Teril, Timonil, Trimonil, Epimaz, Carbama/Carbamaze (in New Zealand), Amizepin( in Poland ), Hermolepsin (Sweden) and Degranol (in South Africa)READ MORE POST

Carbamazepine was discovered by chemist Walter Schindler at J.R. Geigy AG (now part of Novartis) in Basel, Switzerland, in 1953.[2] Schindler then synthesized the drug in 1960, before its anti-epileptic properties had been discovered.
Carbamazepine was first marketed as a drug to treat trigeminal neuralgia in 1962. It has been used as an anticonvulsant in the UK since 1965, but only approved in the U.S. since 1974.
In 1971, Drs. Takezaki and Hanaoka first used carbamazepine to control mania in patients refractory to antipsychotics (lithium was not available in Japan at that time). Dr. Okuma, working independently, did the same thing with success. As they were also epileptologists, they had some familiarity with the anti-aggression effects of this drug. Carbamazepine would be studied for bipolar disorder throughout the 1970s.[3]
[edit]Adverse effects
Carbamazepine is known to render many hormonal contraception products ineffective, due to its action as a cytochrome P450 enzyme inducer, which is the system that metabolizes many oral contraceptives. Carbamazepine causes more cytochrome P450 enzyme to be produced, which hastens removal of the contraceptive from the blood plasma although the clinical significance of this effect is debatable.
Common side effects include drowsiness, headaches and migraines, motor coordination impairment and/or upset stomach. Carbamazepine preparations typically greatly decrease a person's alcohol tolerance.
Less common side effects include cardiac arrhythmias, blurry or double vision and/or the temporary loss of blood cells or platelets and in rare cases can cause aplastic anemia. With normal use, small reductions in white cell count and serum sodium are common, however, in rare cases, the loss of platelets may become life-threatening. This occurs commonly enough that a doctor may recommend frequent blood tests during the first few months of use, followed by three to four tests per year for established patients. In the UK, testing is generally performed much less frequently for long-term carbamazepine patients--typically once per year. Additionally, carbamazepine may exacerbate preexisting cases of hypothyroidism, so yearly thyroid function tests are advisable for persons taking the drug.
There are also reports of an auditory side effect for carbamazepine use, whereby patients perceive sounds about a semitone lower than their actual pitch.[4] Thus, middle C would be heard as the note B3 just below it, etc. This unusual side-effect is usually not noticed by most people, and quickly disappears after the person stops taking carbamazepine.
Oxcarbazepine, a derivative of carbamazepine, reportedly has fewer and less serious side effects.
Carbamazepine may cause Syndrome of inappropriate antidiuretic hormone, since it both increases the release and potentiates the action of ADH (vasopressin).
Carbamazepine may aggravate juvenile myoclonic epilepsy, so it is important to mention any history of jerking, especially in the morning, before starting to take this drug.
Pregnant women taking carbamazepine put their fetuses at increased risk for teratogenic effects. As a result, they should be given folic acid supplementation and undergo prenatal ultrasonography for diagnosis.
In addition, carbamazepine has been linked to serious adverse cognitive anomalies, including EEG slowing[5] and cell apoptosis.[6]
The FDA informed health care professionals that dangerous or even fatal skin reactions (Stevens Johnson syndrome and toxic epidermal necrolysis), that can be caused by carbamazepine therapy, are significantly more common in patients with a particular human leukocyte antigen (HLA) allele, HLA-B*1502. This allele occurs almost exclusively in patients with ancestry across broad areas of Asia, including South Asian Indians. [7] In Europeans a large proportion of sensitivity is associated with HLA-B58.
[edit]Mechanism of action
The mechanism of action of carbamazepine and its derivatives is relatively well understood. Voltage-gated sodium channels are the molecular pores that allow brain cells (neurons) to generate action potentials, the electrical events that allow neurons to communicate over long distances. After the sodium channels open to start the action potential, they inactivate, essentially closing the channel. Carbamazepine stabilizes the inactivated state of sodium channels, meaning that fewer of these channels are available to open, making brain cells less excitable (less likely to fire).
[edit]Interactions
Valproic acid and valnoctamide both interact with carbamazepine, as they inhibit microsomal epoxide hydrolase (mEH), the enzyme responsible for the breakdown of carbamazepine-10,11 epoxide into inactive metabolites.[8] By inhibiting mEH, valproic acid and valnoctamide cause a buildup of the active metabolite, prolonging the effects of carbamazepine and delaying its excretion.
Carbamazepine interacts with multiple drugs and caution should be used in combining other medicines with it. Lower levels of carbamazepine are seen when administrated with phenobarbital, phenytoin (Dilantin), or primidone (Mysoline). Carbamazepine, as CYP 450 inducer, may increase clearance of many drugs, decreasing their blood levels.[9] Drugs that are more rapidly metabolized with carbamazepine include warfarin (Coumadin), phenytoin (Dilantin), theophylline, and valproic acid (Depakote, Depakote ER, Depakene, Depacon).[citation needed] Carbamazepine levels are elevated when taken with erythromycin, cimetidine (Tagamet), propoxyphene (Darvon), and calcium channel blockers.[citation needed] Carbamazepine also increases the metabolism (destruction) of the hormones in birth control pills and can reduce their effectiveness, potentially leading to unexpected pregnancies.REF:WIKIPEDIA
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